It was a promise Matt Perrin wasn't able to keep.
Many people love the holidays because they are a time to make happy memories with loved ones.
Scientists at the Universities of St Andrews and Edinburgh have discovered the functions of the area of the brain in which Alzheimer's begins, offering hope for the development of future treatments.
A common mutation in a key enzyme involved in alcohol metabolism increases damage in cells from patients with Alzheimer's disease and in mice, according to a study by researchers at the Stanford University School of Medicine.
The colour of our eyes or the straightness of our hair is linked to our DNA, but the development of Alzheimer's disease isn't exclusively linked to genetics, suggest recently published findings.
In mouse models of Alzheimer's disease, the investigational drug candidates known as CMS121 and J147 improve memory and slow the degeneration of brain cells. Now, Salk researchers have shown how these compounds can also slow aging in healthy older mice, blocking the damage to brain cells that normally occurs during aging and restoring the levels of specific molecules to those seen in younger brains.
Fresh insights into damaging proteins that build up in the brains of people with Alzheimer's disease could aid the quest for treatments.
An inflammatory marker called sCD14 is related to brain atrophy, cognitive decline and dementia, according to a study of more than 4,700 participants from two large community-based heart studies. The study was published Monday, Dec. 9, in the journal Neurology.
A ketone-supplemented diet may protect neurons from death during the progression of Alzheimer's disease, according to research in mice recently published in JNeurosci.